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Valentina Cenedese
Valentina Cenedese
Dirección de correo verificada de nin.knaw.nl
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Extracellular ATP hydrolysis inhibits synaptic transmission by increasing ph buffering in the synaptic cleft
R Vroman, LJ Klaassen, MHC Howlett, V Cenedese, J Klooster, ...
PLoS biology 12 (5), e1001864, 2014
922014
Anoctamin 2/TMEM16B: a calcium‐activated chloride channel in olfactory transduction
S Pifferi, V Cenedese, A Menini
Experimental physiology 97 (2), 193-199, 2012
722012
The voltage dependence of the TMEM16B/anoctamin2 calcium-activated chloride channel is modified by mutations in the first putative intracellular loop
V Cenedese, G Betto, F Celsi, OL Cherian, S Pifferi, A Menini
Journal of General Physiology 139 (4), 285-294, 2012
482012
Interactions between permeation and gating in the TMEM16B/anoctamin2 calcium-activated chloride channel
G Betto, OL Cherian, S Pifferi, V Cenedese, A Boccaccio, A Menini
Journal of General Physiology 143 (6), 703-718, 2014
422014
Pannexin 1 is critically involved in feedback from horizontal cells to cones
V Cenedese, W De Graaff, T Csikós, M Poovayya, G Zoidl, M Kamermans
Frontiers in molecular neuroscience 10, 403, 2017
212017
Assessment of the Olfactory Function in Italian Patients with Type 3 von Willebrand Disease Caused by a Homozygous 253 Kb Deletion Involving VWF and TMEM16B/ANO2
V Cenedese, M Mezzavilla, A Morgan, R Marino, CP Ettorre, ...
10.1371/journal.pone.0116483, 2015
142015
Interactions between permeation and gating in the TMEM16B/anoctamin2 calcium-activated chloride channel (vol 143, pg 703, 2014)
G Betto, OL Cherian, S Pifferi, V Cenedese, A Boccaccio, A Menini
JOURNAL OF GENERAL PHYSIOLOGY 144 (1), 123-123, 2014
2014
The voltage-dependence of TMEM16B/anoctamin2: the calcium-activated chloride channel in olfactory transduction
V Cenedese
SISSA, 2012
2012
Assessment of the olfactory function in Italian patients with type 3 von Willebrand disease caused by a homozygous 253 Kb deletion involving VWF and TMEM16B/ANO2.
A Menini, V Cenedese
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Artículos 1–9